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TRANSCRIPT
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DIAGNOSTICO DE LAS ARRITMIA
ESTUDIO ELECROFISIOLOGICO
EKGTEST DE ESFUERZO HOLTER
HISTORIA CLINICANADA PUEDE
REMPLAZARLAEXAMINA BIEN A TU
PACIENTEESCUCHA AL PACIENTE QUE EL TE SOPLA EL DX
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ELECTROFISIOLOGIA• El intervalo A-H indica la transmisión del impulso desde
la aurícula alta (catéter auricular) hasta las cercanías del haz de His (par proximal del catéter cabalgando en la válvula tricúspide)
• el par distal de ese catéter registra la deflexión del haz de His, onda H del registro intracavitario.
• Desde esa onda (H) y hasta el inicio de la despolarización ventricular, es el intervalo H-V, es decir, el tiempo que media desde que el estímulo llega al His hasta cuando se despolarizan los ventrículos
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ENFOQUE TERAPEUTICOHC
MONITOR CDEEKG-VIA VENOSA-LABORATORIO
FALLA HEMODINAMICA CARDIOVERSION ELECTRICA
monofásico O BIFASICO100-200-300 JOULES
DX
ESTABILIDAD HEMODINAMICADX
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DIAGNOSTICO DE LA ARRITMIA
ESTUDIO ELECROFISIOLOGICO
EKGTEST DE ESFUERZO HOLTER
HISTORIA CLINICANADA PUEDE REMPLAZARLA
EXAMINA BIEN A TU PACIENTE
ESCUCHA AL PACIENTE QUE EL TE SOPLA EL DX
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ENFOQUE TERAPEUTICOHC
MONITOR CDEEKG-VIA VENOSA-LABORATORIO
FALLA HEMODINAMICA CARDIOVERSION ELECTRICA
monofásico O BIFASICO100-200-300 JOULES
DX
ESTABILIDAD HEMODINAMICADX
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LOCALIZACION ANATOMICA DE LAS ARRITMIAS
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ARRITMIAS SUPRAVENTRICULARES1.-EXTRASISTOLES SUPRAVENTRICULARES
ORIGEN NODULO SINOAURICULAR1.-TAQUICARDIA SINUSAL2.-BRADICARDIA SINUSAL3.-ARRITMIA SINUSAL RESPIRATORIA4.-BLOQUEO SINOAURICULAR(BSA)—TAQUI BRADICARDIA
ORIGEN AURICULAR1.- FIBRILACION AURICULAR2.- FLUTTER O ALETEO AURICULAR3.-RITMO AURICULAR CAOTICO-TAQUICARDIA AURICULAR MULTIFOCAL4.- TAQUICARDIA AURICULAR
NODO AV1.-TAQUICARDIA POR REENTRADA DE LA UNION AV2.-ARRITMIAS MEDIADA POR VIA ACCESORIA
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ARRITMIAS VENTRICULARES1-EXTRASISTOLES2.-TAQUICARDIA VENTRICULAR2a MONOMORFAS2b POLIMORFAS2c TORSION DE PUNTAS3.-ALETEO(FLUTTER VENTRICULAR)4.-FIBRILACION VENTRICULAR5.-RITMO VENTRICULAR AGONICO
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ARRITMIAS FISIOLOGICAS
1)TAQUICARDIA SINUSAL
2)BRADICARDIA SINUSAL
3)ARRITMIA SINUSAL RESPIRATORIA
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TAQUICARDIA SINUSALFC>100,
RESPUESTA FISIOLOGICA AL EJERCICIO ANSIEDAD,EMOCIONES,EMBARAZO
PATOLOGICAS FIEBRE,HIPERTIROIDISMO,ANEMIA FARMACOS
ATROPINA,ANTIASMATICOS-TEOFILINA O
MECANISMO COMPENSADOR A FALLA CARDIACA
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TAQUICARDIA SINUSALorigen supraventriular
una onda P por cada QRSfrecuencia mas de 100
PR normal
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BRADICARDIA SINUSALFC <60
SE PRESENTA EN ATLETAS RESPUESTA VAGAL
FARMACOS (DIGITAL,BETABLOQUEADORES,)
HIPOTIROIDISMO
HIPERTENSION ENDOCRANEANA
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ARRITMIA SINUSALRESPIRATORIA
INSPIRACIONINHIBICION TONOVAGAL PREDOMINA SIMPATICO
RESULTADO TAQUICARDIA
EXPIRACIONPREDOMINA PARASIMPATICO(TONO VAGAL)
RESULTADOBRADICARDIA
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CARACTERISTICAS DE LA FIBRILACION AURICULAR
1. ORIGEN SUPRAVENTRICULAR QRS ANGOSTO2. ONDA P REMPLAZADA POR ONDAS FIBRILATORIAS “f”3. RITMO IRREGULAR TODO RITMO IRREGULAR SIN P ES FIBRILACION
AURICULAR4. FRECUENCIA DE LA ONDA F ENTRE 400 A 600 POR MINUTO 5. RESPUESTA VENTRICULAR RAPIDA -.LENTA O INTERMEDIA
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EXTRASISTOLES COMPLEJO PREMATURO
QUE SE ADELANTASE ORIGINA EN SITIO DIFERENTE
DEL NODO
SINUSAL
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CONCEPTOS• BIGEMINISMO• TRIGEMINISMO• DUPLA• ACOPLAMIENTO• MONOMORFA• POLIMORFA• PAUSA COMPENSADORA• FENOMENO DE R SOBRE T
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EXTRASISTOLIA
SUPRAVENTRICULAR
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EXTRASISTOLE AURICULAR• COMPLEJO PREMATURO(QUE SE ADELANTA)• SE ORIGINA EN SITIO DIFERENTE DEL NODO
SINUSAL MORFOLOGIA DE LA P UN POCO DIFERENTE AUNQUE EL PR ES NORMAL
• QRS ESTRECHO CASI IGUAL AL QRS DE BASE SI HAY BLOQUEO EL QRS ES
ABERRANTE• PUEDE TENER PAUSA COMPENSADORA O
NO
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QRSNORMAL
QRS ANCHO
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EXTRASISTOLE
VENTRICULAR
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EXTASISTOLE VENTRICULARDEFINICION
LATIDO PREMATURO-SE ORIGINAN POR DEBAJO DE LA BIFURCACION DEL HAS DE HIS SON FRECUENTES SOBRETODO EN PACIENTES DE EDAD Y CON CARDIOPATIA
CRITERIOS ELECTROCARDIOGRAFICOS
QRS DEFORME ANCHO >120 MSEG
CASI SIEMPRE PAUSA COMPENSADORA
TRASTORNO DE REPOLARIZACION ONDA T EN SENTIDO CONTRARIO AL QRS
CLINICAMENTE PALPITACION-LATIDO CARDIACO POSTEXTRASISTOLICO
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NO PAUSA COMPENSADORASI PAUSA COMPENSADORA
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EXTRASISTOLE VENTRICULAR
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BIGEMINISMO TRIGEMINISMODUPLETA
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EXTRASISTOLE VENTRICULAR
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BIGEMINISMO-UNIFOCAL-MULTIFOCAL-PAREADA-ACOPLAMIENTO
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FENOMENO DE R SOBRE T
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TRIGEMINISMO-UNIFOCAL
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BIGEMINISMO TRIGEMINISMO PAUSA COMPENSADORA
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LOCALIZACION ANATOMICA DE LAS ARRITMIAS
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ARRITMIAS
DELARRITMIAS
DEL
TEJIDO DE LA
UNION AV
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RITMO DE LA UNION AV P PRE-INTRA Y POST QRS
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EKG UNIÓN AV• PRECEDE AL QRS , NEGATIVA DII-DIII-VF
POSITIVA AVR• PUEDE VISUALIZARSE• NO VISUALIZARSE OCULTA EN EL QRS• VISUALIZARSE DESPUES DEL QRS
PRODUCIENDO UNA MELLA EN EL ST
ONDA P
• NORMAL CON EXCEPCION SI EXISTE PREVIAMENTE BLOQUEOS DE RAMA
• RR CONSTANTE 40-60 QRS
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LOCALIZACION ANATOMICA DE LAS ARRITMIAS
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ARRITMIAS SUPRAVENTRICULARES
PLANOAURICULAR AUSENCIA DE ONDA P
1. FIBRILACION AURICULAR REMPLAZO POR ONDAS f2. FLUTTER AURICULAR REEMPLAZO POR ONDAS F
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FIBRILACION
AURICULAR
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CARACTERISTICAS DE LA FIBRILACION AURICULAR
1. ORIGEN SUPRAVENTRICULAR QRS ANGOSTO2. ONDA P REMPLAZADA POR ONDAS
FIBRILATORIAS “f”3. RITMO IRREGULAR TODO RITMO IRREGULAR
SIN P ES FIBRILACION AURICULAR4. FRECUENCIA DE LA ONDA F ENTRE 400 A 600
POR MINUTO 5. RESPUESTA VENTRICULAR RAPIDA -.LENTA O
INTERMEDIA
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FA es responsable del 15-20% de strokes isquémicos
FA aumenta el riesgo de stroke 4 a 5 veces
FA es un factor de riesgo independiente para la severidad y la recurrencia del stroke
El riesgo de stroke persiste en fibrilados asintomáticos
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CHADSC FALLA CARDIACA 1 PUNTOH HIPERTENSION 1 PUNTOA EDAD >75 AÑOS 1 PUNTOD DIABETES 1 PUNTO S ACV O ICT 2 PUNTOS
CON 2 O MAS PUNTOS EL PACIENTE DEBE ESTAR ANTICOAGULADO CON WARFARINA CON INR
ENTRE 2Y 3
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FLUTTER O
ALETEOAURICULAR
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FLUTTER AURICULAR• ARRITMIA SUPRAVENTRICULAR• FRECUENCIA 300+-50 ONDA F
(SERRUCHO)• F INVERTIDAS EN DII-DIII-VF
• QRS ESTRECHO,REGULAR O IRREGULAR• RELACION 2*1 o 4*1
• MASAJE CAROTIDEO VALOR DIAGNOSTICO NO TERAPEUTICO –AUSCULTAR CAROTIDA LADO DERECHO EN DIESTRO POR 5 A 10
SEGUNDOS
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FLUTTER AURICULAR
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FL A
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OJO•RITMO IRREGULAR SIN P
ES FA•RITMO REGULAR SIN P RITMO DE LA UNION AV
O DE LA ZONA AV
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LOCALIZACION ANATOMICA DE LAS ARRITMIAS
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CONDUCCION AV• ORTODRÓMICA: (orto, sigue el
camino normal; drómica, conducción). conducción anterógrada a través de la
unión AV.• ANTIDRÓMICA: La conducción del
impulso a través de la UAV es en sentido inverso; el impulso sube de
ventrículo a Aurícula.
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HIPERVINCULO
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hipervínculo
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WPW
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TAQUICARDIA POR RENTRADA DE LA UNION AV (TRUAV) TIPICA(LENTA-RAPIDA)
• TAQUICARDIA POR REENTRADA DE LA TRUAV, VARIEDAD ATÍPICA
(RAPIDA-LENTA)
• TAQUICARDIA REENTRANTE AURICULOVENTRICULAR (TRAV)
VIA ACCESORIA
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CONDUCCION AVNORMALMENTE LAS AURICULAS
Y LOS VENTRICULOS ESTAN UNIDOS ELECTRICAMENTE POR EL
NODO AVCUANDO SE UNEN POR OTRAS
VIAS PRODUCEN LOS SINDROMES DE PREEXCITACION
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CONDUCCION AV• ORTODRÓMICA: (orto, sigue el
camino normal; drómica, conducción). conducción anterógrada a través de la
unión AV.• ANTIDRÓMICA: La conducción del
impulso a través de la UAV es en sentido inverso; el impulso sube de
ventrículo a Aurícula.
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TSV POR REENTRADA• Las TSV por reentrada son más
frecuentes en varones, los primeros episodios aparecen entre los 20-40 años, y se asocian a alguna cardiopatía en hasta el 30% de los casos. Esta modalidad de taquicardia por reentrada de la UAV es la más frecuente (>85%), utiliza la vía lenta de modo anterógrado y la vía rápida para la conducción retrógrada.
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TSV POR REENTRADA• Los pacientes que desarrollan este tipo
de arritmia tienen una doble vía nodal fisiológicamente establecida, en la cual una de las vías tiene una velocidad de conducción rápida y período refractario largo (vía beta), la otra vía tiene una velocidad de conducción lenta y período refractario corto (vía alfa).
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DOBLE VIA NODAL FISIOLOGICA
La presencia de doble vía nodal puede demostrarse en hasta el 85% de los pacientes, esta doble vía es un rasgo hereditario que si está en ambos padres, la posibilidad de trasmitirla alcanza el 90%; en ellos se observa una alta conductividad anterógrada por la vía lenta, y retrógrada de la vía rápida; por el contrario, en pacientes que no tienen TRUAV, la vía rápida tiene una escaso o nula conductividad retrógrada, por lo que la presentación de esta arritmia es improbable.
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TAQUICARDIA POR RENTRADA DE LA UNION AV (TRUAV) TIPICA(LENTA-RAPIDA)
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TAQUICARDIA POR RENTRADA DE LA UNION AV (TRUAV) TIPICA(LENTA-RAPIDA)
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KENT AV-JAMES AN-BRECHENMATER AF-MAHAIM NV FV
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TAQUICARDIA REENTRANTE AURICULOVENTRICULAR (TRAV) VIA
ACCESORIA
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Patentes de intervalo QT largo congénito ( niños pescadores)A veces magnificada por causas que prolongan el intervalo QT (diuréticos,
hipokalemia, hipomagnesemia, antidepresivos tricíclicos, macrólidos, dietas líquidas, entre otros).
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El síndrome de Brugada es de probada transmisión genética relacionada con alteraciones en los canales de Na+, los pobladores de países del sudeste
asiático tienen la incidencia de MSC más alta relacionada con este síndrome.
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MIOCARDIOPATIA HIPERTROFICAYAMAGUCHI
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MUERTE SÚBITA• MUERTE SÚBITA CARDIACA (MSC) muerte natural de
causa cardiaca que sucede en la primera hora de iniciados los síntomas. la situación clínica previa a la muerte generalmente es una taquiarritmia (taquicardia ventricular o fibrilación ventricular), muy pocas veces su causa es alguna bradiarritmia (bloqueo AV III, asistolia o la actividad eléctrica sin pulso) presente
• LA MUERTE SÚBITA RESUCITADA es el evento al cual el paciente pudo sobrevivir luego de maniobras de reanimación cardiopulmonar.
• LA MUERTE INSTANTÁNEA se aplica a aquella que acontece sin síntomas premonitorios. , sus causas son una arritmia grave, embolismo pulmonar masivo, ruptura de aneurisma aórtico o un accidente cerebro vascular grave.
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En algunas oportunidades los que desarrollan un episodio de muerte súbita, tuvieron un episodio de pérdida del
estado de conciencia: síncope.
• Así, tal es la importancia del síncope que se asume
que la única diferencia entre el síncope y la
muerte súbita es que en la primera el paciente se
recupera solo
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RITMO
IDIOVENTRICULAR
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TAQUICARDIA
VENTRICULAR
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LOCALIZACION ANATOMICA DE LAS ARRITMIAS
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TV MONOMORFICAFC 120-220,RR REGULARES,QRS ANCHO,FUSION Y CAPTURA E IGUAL EN LA DERIVACION AUSENCIA DE
COMPLEJOS RS EN PRECORDIALES,DISOCIACION AV
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TV POLIMORFICAVARIACION DE LA MORFOLGIA DEL QRS EN UN MISMA
DERIVACION ,FC 150 A 300
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TVCONCORDANCIA POSITIVA
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TV TORSADE DE POINTESCOMPLEJOS QRS QUE CAMBIAN DE AMPLITUD DA LA APARIENCIA DE
TORSION SOBRE UNA LINEA ISOELECTRICA O CAMBIA DE POLARIDAD
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TORSADE POINTES
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TERAPIA ELECTRICA1. Las dos indicaciones de desfibrilación son la FV y la
taquicardia ventricular sin pulso (52), en estos casos la dosis inicial será 200 J en los desfibriladores monofásicos (los más comúnmente encontrados en las salas de urgencias de nuestro hospitales) 150 J en los bifásicos si no hay mejoría del ritmo inicial se usará una segunda descarga El procedimiento es doloroso debe administrarse anestesia comúnmente con midazolam y fentanyl o propofol
2. La principal indicación de cardioversión eléctrica es la taquiarritmia, ya sea ventricular o supra ventricular, con inestabilidad hemodinámica . Esta inestabilidad es definida por la presencia de hipotensión sintomática, choque, edema pulmonar o síndrome coronario agudo
3. Los cardiodesfibriladores implantados ha surgido gran investigación en esta terapia haciéndola cada vez más segura y efectiva.
4. Marcapasos
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TAQUICARDIA VENTRICULARSOSTENIDA Y NO SOSTENIDA
TV SOSTENIDA
DURACION >30”
PRODUCE COLAPSO
HEMODINAMICO
TV NO SOSTENIDA
DURACION<30”
NO PRODUCE COLAPSO
HEMODINAMICO
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TORSADE POINTES
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Todas las arritmias video
todas las arritmias.mp4
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