Bibliography of hypertrophy - Scalrovsky

1

Fijensen en la izquierda muestra las presiones desarrolladas en ela ratas

con hipertrofia, la hipertrofia disminuye la tension intramural ,mientras

Que no dessarrollan hipertension hay una alta tension intramural

Ley de Laplace

Kelly SHJ et al clonocall exp phasrmacilogy physiology 33.642;2006

Prepado de Langerdof en un raton , registra los potenciales de epicardio y endocardio y un globo intracavitaio r

Note que que el potencial de accion del endocardio se acorta cion el aumento de la pre4sion intracavitaria ,pero no hay cambios en el potencial de accion del epicardio

end

epi

Molecular biology could explain the pattern of high voltage and fast conduction of R waves in left ventricle hypertrophy

The high R wave in v4-5 in apical remodeling indicates the addition of sarcomeres in the longitudinal axis

2004

Conclusions androgen receptors In cardiac myocytes from multiple

species, including normal men and women,in a context that permits the

cardiac phenotype and produce hypertrophy by direct receptors-

specific mechanism for therapeuticalor ilicit use of androgen in men

REModelacion tioca de un hipertensoCon remodelacion fibrotica sin hypertrofia muscular de alto riego para arritmias

atriales y ventricularesל

a)Una tipica remodelacionhypertrofia ventricular musculat

Spinale F, Physiol Rev 2007

Spinal;e G Physiological Review 2007

Metalloproteinases(MMP) regulate collagen I concentration in extra cellular matrix.

The collagen turnover is about 90 days.

Tissue Inhibitor Metalloproteinase (TIMP) regulates the concentration of MMP

Lombardi R et al, Circulation 2003Lombardi R et al, Circulation 2003

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Myocardial Matrix Remodeling and the Matrix Metalloproteinases: Influence on Cardiac Form and Function

Francis G. Spinale

Physiol. Rev. 87: 1285-1342, 2007; doi:10.1152/physrev.00012.2007 0031-9333/07 $18.00

.Part I: signal transduction and basic electrophysiological mechanismsAndreas Goette eurpace1, and

Uwe Lendeckel2 + Author Affiliations

1Division of Cardiology, University Hospital Magdeburg, Leipzigerstr. 44, Magdeburg 39120, Germany

2Institute of Experimental Internal Medicine, University Hospital Magdeburg, Magdeburg 39120, Germany

*Corresponding author. Tel: +49 391 6713225; fax: +49 391 671 3202.E-mail address :andreas.goette@medizin.uni-magdeburg.de

Received October 17, 2007 .Accepted November 28, 2007 .

Next SectionAbstract

In the recent years, a tremendous amount has been learned about the pathophysiology and molecular biology of the cardiac angiotensin II system. Interestingly, the angiotensin II appears to have several effects on cardiac

electrophysiology, which have not been fully appreciated so far. Therefore, this review will highlight on the basic knowledge of signal transduction and

electrophysiological effects of angiotensin II, which may have an impact on the occurrence of cardiac arrhythmia .

Key wordsAngiotensin Arrhythmias

Fibrillati